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To reconcile the findings that both GIPR antagonists and GIPRAs reduce body weight, it is possible that physiological GIPR activity plays a permissive role in adipose tissue synthesis, while pharmacologically enhanced GIPR activity may reduce body weight through a different mechanism in the brain

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Relevant brain areas implicated in GLP-1 control of body weight and food intake include the the hypothalamic arcuate nucleus 57,58,59 and the hindbrain DVC 59,60 , hence the same brain regions that are targeted by long-acting GIPR agonists 15,20,21